best sugar substitute for diabetics

🔥+ best sugar substitute for diabetics 08 Aug 2020 Many of the symptoms of diabetes can be subtle. Maybe you're thirstier or more hungry than usual. Perhaps you've noticed patches of darker ...

best sugar substitute for diabetics It is related to diabetic ketoacidosis (DKA), another complication of diabetes more often (but not exclusively) encountered in people with type 1 diabetes; they are ...

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Maturity onset diabetes of the young (MODY) is a heterogeneous group of monogenic disorders distinct from type 1 (insulin-dependent) and type 2 (noninsulin-dependent) diabetes (1,2).

  • there is β cell dysfunction characterised by non-ketotic diabetes and absence of pancreatic auto-antibodies (3)

The for 1 last update 08 Aug 2020 condition was first described by Tattersall in 1974–1975, a familial form of non-insulin dependent diabetes which showed autosomal dominant inheritance which presented before the age of 25 years (1,2). The molecular genetic basis of MODY was subsequently recognised in the 1990s and mutations in 10 different genes have been identified so far (2). The condition was first described by Tattersall in 1974–1975, a familial form of non-insulin dependent diabetes which showed autosomal dominant inheritance which presented before the age of 25 years (1,2). The molecular genetic basis of MODY was subsequently recognised in the 1990s and mutations in 10 different genes have been identified so far (2).

Prevalence of MODY is unknown but the minimum UK population prevalence of all MODY subtypes is estimated to be 68-108 cases per million.

  • the prevalence of MODY subtypes differ from country to country
    • in countries such as  France, Spain, and Italy where glucose testing of asymptomatic people is done routinely, glucokinase (GCK) mutations are commonly diagnosed while in countries where random blood glucose tests are seldom done  hepatocyte nuclear factor 1α (HNF1A) is commonly daiagnosed.
  • in the UK, most common causes of mutations of MODY are
    • glucokinase (GCK) - 32%
    • hepatocyte nuclear factor 1α (HNF1A) - 52%
    • hepatocyte nuclear factor 4α (HNF4A) - 10%  (3)

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